Most hair fall after pregnancy is telogen effluvium and resolves without treatment. Hypothyroidism makes shedding worse: in a study of 500 women with telogen effluvium, those with hypothyroidism had significantly more severe hair loss (Bin Dayel et al., Medicine, 2024). Postpartum thyroiditis affects an estimated 5 to 10% of women in the year after childbirth, and its hypothyroid phase lines up with the peak shedding window. Hypothyroidism is markedly more common in Indian women than the global average, which is why a thyroid test is worth considering when postpartum shedding is severe or persistent. Topical scalp care can run alongside testing, but it does not replace treating a thyroid problem.
Quick Answer: Postpartum hair fall is usually telogen effluvium, a temporary shedding that begins about two to four months after delivery and settles on its own within six to twelve months. It becomes worth investigating when it is severe, drags on past six months, or arrives alongside other symptoms, because hypothyroidism and postpartum thyroiditis are common, treatable causes that often go undiagnosed. This matters more in India than global figures suggest: a 2021 meta-analysis of 61 Indian studies found hypothyroidism in about 11% of pregnant women, against 1.5 to 4% reported worldwide (Yadav et al., Journal of Thyroid Research, 2021). If your shedding is heavy or will not slow down, a simple TSH and free T4 blood test is the most useful next step.
Is Hair Fall After Pregnancy Normal, or a Sign of a Thyroid Problem?
Most hair fall after pregnancy is normal. Postpartum telogen effluvium is diffuse hair shedding caused by the hormonal shift after delivery: as progesterone falls and prolactin rises, a large share of hair follicles move prematurely from the growth (anagen) phase into the resting (telogen) phase, and those hairs are shed a few months later (Galal et al., Journal of Clinical and Aesthetic Dermatology, 2024). It is the body correcting the unusually thick hair many women enjoy during pregnancy, when those same hormones keep follicles in the growth phase for longer.
Telogen effluvium is classed as acute when it lasts under six months, which covers most postpartum cases. Hair regrowth is expected once the trigger has passed, so the standard reassurance that it will resolve is true for the majority of women.
The reassurance becomes incomplete when shedding is severe, lasts beyond six months, or comes with other symptoms. Telogen effluvium is a diagnosis of exclusion, which means it is the label applied once other causes have been ruled out. When the shedding is heavy or stubborn, the cause worth ruling out first is the thyroid, because thyroid problems are both common after pregnancy and straightforward to treat.
Hypothyroidism is far more common in Indian women than global averages imply, and that changes the math on testing. A 2021 meta-analysis of 61 Indian observational studies put the pooled prevalence of hypothyroidism in pregnant women at 11.07%, compared with the 1.5 to 4% reported in other countries (Yadav et al., Journal of Thyroid Research, 2021). A separate epidemiological study across 11 cities in 9 Indian states found hypothyroidism in 13.13% of pregnant women in the first trimester, most of it subclinical (Dhanwal et al., Indian Journal of Endocrinology and Metabolism, 2016).
The pattern holds in the wider adult population. A 2013 study across eight Indian cities found hypothyroidism in 10.95% of adults, roughly 1 in 10, with the condition significantly more common in women (Unnikrishnan et al., Indian Journal of Endocrinology and Metabolism, 2013). In that study, 3.47% of all adults had hypothyroidism that had never been detected, and anti-thyroid peroxidase (anti-TPO) antibodies, a marker of thyroid autoimmunity, were found in 21.85% of participants.
Two facts from that data matter for new mothers. A meaningful share of Indian hypothyroidism is undiagnosed, and roughly one in five Indian adults carries the autoimmunity marker that predisposes a woman to thyroid trouble after pregnancy. When an Indian woman experiences heavy postpartum shedding, a thyroid problem is a realistic and testable explanation, not a remote one.
Hypothyroidism does not just cause hair loss, it intensifies it. Thyroid hormones help regulate the hair follicle cycle, and when levels are low the follicles are pushed into and held in the resting phase, prolonging shedding and slowing the return to growth.
The clearest evidence comes from a 2024 retrospective study of 500 women with telogen effluvium who had thyroid testing within three months of diagnosis (Bin Dayel et al., Medicine, 2024). Of those women, 248 (49.6%) had normal thyroid function, 150 (30%) had hypothyroidism, and 102 (20.4%) had hyperthyroidism. The hypothyroid group had a significantly higher mean Severity of Alopecia Tool (SALT) score, meaning their hair loss was measurably worse than that of women with normal thyroid function.
That study was conducted in Saudi Arabia and looked at telogen effluvium from any cause, not postpartum shedding specifically, so it does not prove that postpartum hair fall is usually thyroid-driven. What it does establish is a clear and treatable pattern: when telogen effluvium is severe, hypothyroidism is the dysfunction most likely to be behind it. For a new mother, severe shedding is itself a reason to check thyroid function.
Postpartum thyroiditis is a destructive autoimmune inflammation of the thyroid gland that develops within the first year after childbirth in women who had normal thyroid function before pregnancy. It affects an estimated 5 to 10% of women in the postpartum period, with reviews reporting wider ranges across different regions and diagnostic criteria (StatPearls, Postpartum Thyroiditis, 2025).
Postpartum thyroiditis is missed so often because it hides behind ordinary new-parent life. It typically moves through three phases, and the symptoms of each are easily mistaken for the normal exhaustion and emotional flux after a baby arrives.
Thyrotoxic phase, around one to four months after delivery. Thyroid hormone levels run high, producing anxiety, palpitations, insomnia, irritability and weight loss. These are routinely written off as the stress of a newborn (American Thyroid Association, Postpartum Thyroiditis).
Hypothyroid phase, around three to six months after delivery, sometimes later. Hormone levels fall, producing fatigue, weight gain, low mood, cold intolerance, constipation and hair loss. This phase overlaps almost exactly with the peak window for postpartum hair shedding, which is why so much thyroid-driven shedding is mislabelled as ordinary telogen effluvium.
Recovery phase, usually within twelve months. Most women return to normal thyroid function by the end of the first year (Endotext, Postpartum Thyroiditis, 2018).
Recovery is the common outcome, but it is not guaranteed. Estimates of how many women with postpartum thyroiditis go on to permanent hypothyroidism range from about 20 to 50%, and one review put the figure at roughly 25% within the following ten years (StatPearls, 2025). Because of that long-term risk, a woman diagnosed with postpartum thyroiditis should have her thyroid monitored well beyond the first year.
Get a thyroid test if your postpartum shedding is severe, is still heavy beyond six months, or comes with other symptoms of thyroid dysfunction. Routine postpartum shedding that is slowing down on its own does not need investigation. The table below shows the difference.
|
Feature |
Typical postpartum telogen effluvium |
Worth a thyroid check |
|
Timing |
Starts 2 to 4 months after delivery, eases by 6 to 12 months |
Still heavy beyond 6 months, or worsening rather than easing |
|
Pattern |
Diffuse, even thinning across the scalp, gradually slowing |
Severe shedding, or visible scalp showing through |
|
Other symptoms |
None beyond shedding |
Fatigue, weight change, low mood, cold intolerance, constipation, palpitations |
|
History |
No prior thyroid or hair-loss history |
Family history of thyroid disease, or hair thinning that predates pregnancy |
The first-line test is straightforward. A TSH (thyroid-stimulating hormone) and free T4 blood test screens for both hypothyroidism and postpartum thyroiditis. If those results are abnormal, or if there is a family history of autoimmune thyroid disease, a doctor may add anti-TPO and anti-thyroglobulin antibody tests to confirm autoimmunity.
A full workup for stubborn postpartum shedding usually also looks beyond the thyroid. A complete blood count with serum ferritin checks for iron deficiency, which is common after pregnancy, and vitamin D and B12 levels are often tested too, since nutritional deficiencies can drive shedding in parallel. A dermatologist can use trichoscopy, a non-invasive magnified scalp examination, to tell telogen effluvium apart from female-pattern hair loss.
Mylo Clinic offers gynaecologist consultations, which is a practical entry point for a postpartum thyroid workup. A consulting doctor can order TSH and free T4 testing, interpret the results in the context of your delivery and symptoms, and refer you to an endocrinologist or dermatologist if needed.
Hair loss that began before pregnancy is a clinically useful signal, and it points in two directions. The first is pattern hair loss. Pregnancy hormones can temporarily mask female-pattern hair loss, also called androgenetic alopecia, and the shedding after delivery can unmask thinning that was already present. In a 2024 study of 200 women evaluated at a dermatology clinic for postpartum hair loss, only 9.5% had telogen effluvium alone, while 56% had telogen effluvium together with androgenetic alopecia (Galal et al., Journal of Clinical and Aesthetic Dermatology, 2024).
That figure comes from a clinic population, women who sought help for their hair, so it should not be read as the rate across all new mothers. The useful takeaway is narrower and still important: among women whose postpartum shedding is troubling enough to warrant evaluation, pure telogen effluvium is the minority, and a second diagnosis is common. If you had thinning hair before pregnancy, mention it to your doctor, because it changes what the shedding is likely to be.
The second direction is the thyroid. Subclinical hypothyroidism that was mild or unnoticed before pregnancy can become overt afterwards, and thyroid autoimmunity present in pregnancy strongly predicts trouble after delivery. Between one-third and one-half of women who carry thyroid peroxidase antibodies during pregnancy go on to develop postpartum thyroiditis (Autoimmune Abnormalities of Postpartum Thyroid Diseases, 2017). If you have ever had a borderline thyroid result or a family history of thyroid disease, that history is worth raising at your postpartum checkup.
Topical scalp care can run alongside a medical workup, but it is important to be clear about what it does and does not do. A scalp oil supports the condition and manageability of the hair you have and makes a calming care routine possible. It does not treat an underlying thyroid problem. If hypothyroidism is driving the shedding, the cause has to be corrected medically, usually with thyroid hormone replacement prescribed by a doctor, before hair recovery can fully follow.
Within that honest frame, the botanical ingredients in a product like Mylo Onion Hair Oil, a Pre and Post Pregnancy Hairfall Oil formulated with Onion Seed Oil, Amla, Bhringraj, Ylang Ylang, Methi and Vitamins A and D3, each have a specific rationale worth understanding rather than just listing.
Bhringraj (Eclipta alba) is the herb Ayurveda calls Keshraj, the King of Hair, and it has the strongest preclinical evidence of the group. In a published study, a methanol extract of Eclipta alba pushed resting follicles back into their growth phase in mice (Datta et al., Journal of Ethnopharmacology, 2009), and in an earlier study, extracts of the plant roughly halved the time to hair-growth initiation in rats, comparing favourably with 2% minoxidil (Roy, Thakur and Dixit, Archives of Dermatological Research, 2008). This evidence is from animal models, not human trials, but it explains why Bhringraj is the central hair herb in Ayurvedic formulations.
Amla (Phyllanthus emblica), the Indian gooseberry, is one of the richest natural sources of vitamin C and antioxidants, which help protect the scalp environment from oxidative stress. Amla is also a documented inhibitor of 5-alpha-reductase, the enzyme that drives pattern hair loss (Kumar et al., Journal of Ethnopharmacology, 2012). That mechanism is most relevant to androgenetic alopecia, so Amla is best understood as supporting follicle and scalp health rather than as a treatment for telogen effluvium.
Methi (fenugreek) has a long traditional use for hair in India and is valued for its high galactomannan content, which conditions the hair shaft and improves slip, reducing breakage when combing the fragile hair of the postpartum months.
Onion Seed Oil is included for the sulphur compounds and quercetin that have made onion a popular hair ingredient. It is worth being precise here: the often-cited 2002 onion-juice trial was conducted in patients with alopecia areata, an autoimmune patchy hair loss, and not in telogen effluvium (Sharquie and Al-Obaidi, Journal of Dermatology, 2002). Its results should not be read as proof that onion treats postpartum shedding. Onion seed oil belongs in this formula as a traditional scalp-care ingredient, not as a cure.
For a fuller wash-and-care routine alongside the oil, the Onion Hair Shampoo is the natural complement, pairing the oil's pre-wash conditioning with a gentle daily cleanser. Used this way, a scalp oil and gentle scalp massage are a reasonable, calming part of the postpartum months. The decisive step for severe or persistent shedding remains the blood test.
Is hair fall after pregnancy related to the thyroid? Sometimes, and it is worth checking when shedding is severe. Most postpartum hair fall is telogen effluvium caused by the normal hormonal shift after delivery, and it resolves on its own. But hypothyroidism intensifies shedding: in a 2024 study of 500 women with telogen effluvium, those with hypothyroidism had significantly more severe hair loss than women with normal thyroid function (Bin Dayel et al., Medicine, 2024). Postpartum thyroiditis, which affects an estimated 5 to 10% of women after childbirth, has a hypothyroid phase that overlaps the peak shedding window. If your shedding is heavy or persistent, a thyroid test is a sensible next step.
When should I get a thyroid test for postpartum hair loss? Get tested if the shedding is severe, is still heavy beyond six months postpartum, or comes with symptoms such as fatigue, weight change, low mood, cold intolerance, constipation or palpitations. The first-line test is a TSH and free T4 blood test, which screens for both hypothyroidism and postpartum thyroiditis. If there is a family history of autoimmune thyroid disease, a doctor may add anti-TPO antibody testing. Routine shedding that is gradually slowing on its own does not need investigation.
How common is hypothyroidism in pregnant women in India? It is far more common in India than in most other countries. A 2021 meta-analysis of 61 Indian studies found a pooled hypothyroidism prevalence of about 11% in pregnant women, against 1.5 to 4% reported worldwide (Yadav et al., Journal of Thyroid Research, 2021). A separate study across 11 Indian cities in 9 states found 13.13% in the first trimester, most of it subclinical (Dhanwal et al., 2016). This higher background rate is the main reason thyroid testing is worth considering for Indian women with troubling postpartum shedding.
Does postpartum thyroiditis go away on its own? Usually, but not always. Most women with postpartum thyroiditis return to normal thyroid function within twelve months of delivery. However, an estimated 20 to 50% develop permanent hypothyroidism, and one review found that roughly 25% do so within the following ten years (StatPearls, 2025). Because of that long-term risk, women diagnosed with postpartum thyroiditis should have their thyroid monitored beyond the first year, even after symptoms settle.
Can I use hair oil if my hair fall is caused by a thyroid problem? Yes, but understand its role. A scalp oil supports the condition and manageability of your hair and makes a gentle care routine possible, and it can be used alongside medical treatment. It does not correct a thyroid problem. If hypothyroidism is driving the shedding, the thyroid itself has to be treated, usually with prescribed thyroid hormone, before hair recovery can fully follow. Topical care is a companion to that treatment, not a substitute for it.
I had hair thinning before pregnancy. Does that mean something? It is a useful signal worth mentioning to your doctor. Pregnancy hormones can temporarily mask female-pattern hair loss, and postpartum shedding can unmask thinning that was already there. Among women evaluated at a clinic for postpartum hair loss, 56% had telogen effluvium together with androgenetic alopecia (Galal et al., 2024). Pre-pregnancy thinning can also reflect a mild thyroid problem that becomes more obvious after delivery. Either way, hair loss that predates your pregnancy changes what the current shedding is likely to be, so your doctor should know about it.
How long does postpartum hair fall normally last? Postpartum telogen effluvium usually begins two to four months after delivery and eases within six to twelve months as follicles return to their growth phase. Shedding that is still heavy beyond six months, or that is worsening rather than slowing, is no longer following the typical pattern and warrants a check, starting with thyroid and iron testing.
Galal SA, El-Sayed SK, Henidy MMH. Postpartum Telogen Effluvium Unmasking Additional Latent Hair Loss Disorders. Journal of Clinical and Aesthetic Dermatology. 2024;17(5):15-22. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC11210592/
Bin Dayel S, Hussein RS, Atia T, Abahussein O, Al Yahya RS, Elsayed SH. Is thyroid dysfunction a common cause of telogen effluvium? A retrospective study. Medicine (Baltimore). 2024;103(1):e36803. https://journals.lww.com/md-journal/fulltext/2024/01050/is_thyroid_dysfunction_a_common_cause_of_telogen.51.aspx
Yadav V, Dabar D, Goel AD, Bairwa M, Sood A, Prasad P, Agarwal SS, Nandeshwar S. Prevalence of Hypothyroidism in Pregnant Women in India: A Meta-Analysis of Observational Studies. Journal of Thyroid Research. 2021;2021:5515831. https://onlinelibrary.wiley.com/doi/10.1155/2021/5515831
Dhanwal DK, Bajaj S, Rajput R, et al. Prevalence of hypothyroidism in pregnancy: An epidemiological study from 11 cities in 9 states of India. Indian Journal of Endocrinology and Metabolism. 2016;20(3):387-390. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4855968/
Unnikrishnan AG, Kalra S, Sahay RK, Bantwal G, John M, Tewari N. Prevalence of hypothyroidism in adults: An epidemiological study in eight cities of India. Indian Journal of Endocrinology and Metabolism. 2013;17(4):647-652. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3743364/
Postpartum Thyroiditis. StatPearls, NCBI Bookshelf. Updated 2025. https://www.ncbi.nlm.nih.gov/books/NBK557646/
Stagnaro-Green A. Postpartum Thyroiditis. Endotext, NCBI Bookshelf. 2018. https://www.ncbi.nlm.nih.gov/books/NBK279115/
American Thyroid Association. Postpartum Thyroiditis patient resource. https://www.thyroid.org/postpartum-thyroiditis/
Di Bari F, Granese R, Le Donne M, Vita R, Benvenga S. Autoimmune Abnormalities of Postpartum Thyroid Diseases. Frontiers in Endocrinology. 2017;8:166. https://www.frontiersin.org/articles/10.3389/fendo.2017.00166/full
Datta K, Singh AT, Mukherjee A, Bhat B, Ramesh B, Burman AC. Eclipta alba extract with potential for hair growth promoting activity. Journal of Ethnopharmacology. 2009;124(3):450-456. https://pubmed.ncbi.nlm.nih.gov/19481595/
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Kumar N, Rungseevijitprapa W, Narkkhong NA, Suttajit M, Chaiyasut C. 5-alpha-reductase inhibition and hair growth promotion of some Thai plants traditionally used for hair treatment. Journal of Ethnopharmacology. 2012;139(3):765-771. https://pubmed.ncbi.nlm.nih.gov/22178180/
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This content is for informational purposes only and should not replace professional medical advice. Consult with a physician or other health care professional if you have any concerns or questions about your health. If you rely on the information provided here, you do so solely at your own risk.

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